第三篇 IQ-gene In the angry debate over how much of IQ comes from the genes that children inherit from parents and how much comes from experiences, one little fact gets overlooked: no one has identified any genes (other than those that cause retardation) that affect intelligence. So researchers led by Robert Plomin of London’s Institute of Psychiatry decided to look for some. They figured that if you want to find a "smart gene", you should look in smart kids. They therefore examined the DNA of students like those who are so bright that they take college entrance exams four years early--and still score at Princeton--caliber levels. The scientists found what they sought. "We have," says Plomin, "the first specific gene ever associated with general intelligence." Plomin’s colleagues drew blood from two groups of 51 children each, all 6 to 15 years old and living in six counties around Cleveland. In one group, the average IQ is 103. All the children are white. Isolating the blood cells, the researchers then examined each child’s chromosome 6 of the 37 landmarks on chromosome 6 that the researchers looked for, one jumped out: a form of gene called IGF2R occurred in twice as many children in the high-IQ group as in the average group--32 percent versus 16 percent. The study, in the May issue of the journal Psychological Science, concludes that it is this form of the IGF2R gene that contributes to intelligence. Some geneticists see major problems with the IQ-gene study. One is the possibility that Plomin’s group fell for "chopsticks fallacy". Geneticists might think they’ve found a gene for chopsticks flexibility, but all they’ve really found is a gene more common in Asians than, say, Africans. Similarly, Plomin’s IQ gene might simply be one that is more common in groups mat emphasize academic achievement. "What is the gene that they’ve found reflects ethnicity" asks geneticist Andrew Feinberg of Johns Hopkins University. "That alone might explain the link to intelligence, since IQ tests are known for being culturally sensitive and affected by a child’s environment. "And Neil Risch of Standford University points out that if you look for 37 genes on a chromosome, as the researchers did, and find that one is more common in smarter kids, that might reflect pure chance rather than a causal link between the gene and intelligence. Warns Feinberg, "I would take these findings with a whole box of salt." What does "some" in the second sentence of paragraph one stands for
A. Parents.
B. Children.
C. Experiences.
D. Genes.
请分析吕某的行为性质的认定和犯罪形态。 1.定罪:吕某架设电线时,用手去碰触包扎的地方,觉得没有电麻的感觉,就轻率地认为不会出事故。吕某的这种已经预见自己的行为可能发生危害社会的结果却轻信能够避免,以致发生这种结果的行为,完全符合过失犯罪的特征。吕某因过失行为致人死亡,构成过失致人死亡罪。 吕某不构成故意杀人罪。因为吕某并没有预见到自己的行为会发生危害社会的后果,更谈不上希望或放任危害结果的发生,而且危害结果的发生显然是违背吕某的意思的。 吕某不构成过失以危险方法危害公共安全罪,对于吕某应当定过失致人死亡罪。因为吕某为了生活用电架设电线,主观上不是为了加害于人,而且在人迹罕至的地方架设电线,客观上也不足以危害公共安全,因此吕某的行为不构成过失以危险方法危害公共安全罪。 2.吕某是过失犯罪,吕某见状帮助受害人进行人工呼吸,并送往医院抢救不构成犯罪中止。犯罪中止是故意犯罪的未完成形态,在犯罪过程中,自动放弃犯罪或者自动有效地防止犯罪结果发生的构成犯罪中止,吕某并无故意犯罪,不构成犯罪中止。 3.吕某不构成累犯,因为其前罪是危害国家安全的犯罪,但后罪是过失致人死亡的犯罪,既不构成一般累犯,也不构成危害国家安全犯罪的特别累犯。
在星期六,张山建议李思下周不工作陪他到某个山区旅游。李思拒绝了,自称他既支付不起旅游费用又得因为请假而被扣工资。然而,费用很明显不是李思不愿陪张山到那个山区旅游的真正原因,因为每次张山邀请他参加一个事先没有安排的旅行时,不管张山计划上哪儿,李思都给出同样的理由。 上面的推论最易受到下面哪一项的攻击
A. 它企图通过把注意力集中在李思的行为上从而预防张山的行为受攻击。
B. 它没有证实张山和李思一样支付不起一个事先没有安排的假期旅行的费用。
C. 它忽视了这种可能,即不像张山那样,李思喜欢一个早就计划好的假期。
D. 它假定如果李思所说的原因不是他的唯一原因,那么这个原因对李思来说根本就不是真正的原因。
E. 它没有调查李思的行为可以被他所给出的原因所充分解释的可能性。
阅读下面这篇短文,短文后列出了7个句子,请根据短文的内容对每个句子作出判断。如果该句提供的是正确信息,请选择A项:如果该句提供的错误信息,请选择B项:如果该句的信息文章中没有提及,请选择C项。 Tanning Parlors Take Heat People who seek a glamorous tan through sun lamps may double their risk of developing Common types of skin cancer, according to a new study that found the risk was highest for those who start at a young age. The study, appearing in the latest issue of Journal of the US National Cancer Institute, concluded that people who use tanning devices were 1.5 to 2.5 times more likely to have common kinds of skin cancer than were people who did not use the devices. The study confirmed what doctors have long suspected-that sun lamp use increases the risk of basal cell and squamous cell skin cancers, said Margaret R. Karagas, first author of the study. Either going to the tanning parlor, or getting an infrequent sunburn can seriously damage the skin, said Dr. James Spencer, vice chairman of the department of dermatology at Mount Hospital, but the small day-to-day exposure is worse for the skin in the long run. Joseph A. Levy, vice president of the International Smart Tan Network, however, said occasional sunburn "is a risk factor in all forms of skin cancer and intermittent sunburn is what the tanning industry is trying to stop. ’ In the study, Karagas and her colleagues interviewed 603 basal cell skin cancer patients and 293 with squamous cell skin cancer. They talked to 540 control subjects who did not have either type of skin cancer. About 1 million Americans are diagnosed annually with skin cancer. Among those skin cancer patients, about 80 percent are with basal cell skin cancer, 16 percent, with squamous cell skin cancer and 4 percent with melanoma-the most serious form of skin cancers. Back to the interviewed patients, 190 reported that they had used tanning devices at some time. In the control groups, only 75 had used such devices. Karagas said a statistical analysis shows that those who used tanning equipment were 2.5 times more likely to get squamous cell skin cancer than those who had not used the devices. For basal cell cancer, the risk was 1.5 times greater. The risk was highest for those who first used the tanning devices before the age of 20, said Karagas. For this group, the squamous cell cancer risk was 3.6 times greater than that of the controls while the basal cell cancer risk was 1.3 times greater. Doctor James Spencer’s argument implies that in the long run, getting all infrequent sunburn is worse than the small day-to-day exposure.
A. Right
B. Wrong
C. Not ment